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J Bone Joint Surg 70: 1402–1403 Linscheid RL (1965) Injuries to radial nerve at the wrist purchase accutane 10 mg. Arch Surg 91: 942–946 Marmor L buy accutane 10mg free shipping, Lawrence JF 10 mg accutane fast delivery, Dubois EL (1967) Posterior interosseus nerve palsy due to rheumatoid arthritis effective accutane 40 mg. J Bone Joint Surg Am 49 (381): 383 Spinner M, Freundlich BD (1968) Posterior interosseus nerve palsy as a complication of Monteggia fractures in children. Clin Orthop 58: 141–145 Sturzenegger M (1991) Die Radialisparesen. Ner- venarzt 62: 722–729 Wartenberg R (1932) Cheiralgia paresthetica: Neuritis des Ramus superficialis Nervi radialis. Z Neurol Psychiatr 141: 145–155 173 Digital nerves of the hand Genetic testing NCV/EMG Laboratory Imaging Biopsy (+) + – Sensory loss in the fingers Symptoms Tinel’s sign, callus, local swelling Signs Trauma Causes Joint abnormalities: mucous cyst from arthritis, osteophytes Mechanical trauma: scissors, bowlers thumb, “mouse neuropathy”, nylon shopping bags Miscellaneous: Diabetes Leprosy Rheumatoid arthritis Vasculitis Musicians: instrument, bow Nerve tumors, Schwannoma Tendon sheath pathology: Cysts Giant cell tumors Rheumatoid tenosynovitis Trauma: Blunt trauma digit and palm Chronic external compression Fractures Lacerations NCV Diagnosis MRI Conservative treatment Therapy Surgical procedures rarely necessary Dawson DM, Hallet M, Wilbourn AJ (1999) Digital nerve entrapment in the hand. In: Reference Dawson DM, Hallet M, Wilbourn AJ (eds) Entrapment neuropathies. Lippincott Raven, Philadelphia, pp 251–263 175 Mononeuropathies: trunk 177 Phrenic nerve Genetic testing NCV/EMG Laboratory Imaging Biopsy Pulmonary function + + – + tests NCV X ray EMG of the Ultrasound diaphragm of diaphragm Fig. Phrenic nerve is in the vicinity of the pericardium. C Expiration Anatomy The phrenic nerve fibers are from C3, 4, and 5. The connection with C3 may be via the inferior ansa cervicalis (cervical plexus). The nerve travels over the anterior scalenus muscle, dorsal to the internal jugular vein, and crosses the dome of the pleura to reach the anterior mediastinum. On the right side, it is positioned next to the superior vena cava and near the right atrium. After transversing the diaphragm, the phrenicoabdominal branches supply the peritoneum of the diaphragm, liver, gall bladder and pancreas. Symptoms Unilateral lesion: mild dyspnea, or asymptomatic. Bilateral lesions: age dependent, with babies and small children developing respiratory problems. Newborns with bilateral lesions require ventilation. Adults are easily dyspneic, particularly upon exertion, and unable to lie in a supine position. Causes Birth trauma (with or without associated brachial plexus lesions) Idiopathic Polyneuropathies (AIDP, critical illness, multifocal neuropathy with conduction block) Neuralgic amyotrophy Frequent sites of lesion Chest: Intrathoracic malignant tumors Chest operations (intraoperative mechanical or local cooling) Neck wounds Traction, with upper trunk of brachial plexus damage Chest radiograph 179 Clinically: respiration, ability to recline supine (Fig. Therapy Trauma cases can be considered for surgical repair (re-innervation may reach related muscles of the upper extremity, such that breathing discharges can be seen in EMG). Adults: unilateral lesions may be compensated, but bilateral lesions may require nighttime respiratory support. Bolton CF, Chen R, Wijdicks EFM, Zifko UA (2004) Neurology of breathing. Butterworth References Heinemann, Elsevier Inc (USA) Cavaletti G (1998) Rapidly progressive multifocal motor neuropathy with phrenic nerve paralysis; effect of nocturnal assisted ventilation. J Neurol 245: 613–616 Chen ZY, Xu JG, Shen LY, et al (2001) Phrenic nerve conduction study in patients with traumatic brachial plexus palsy. Muscle Nerve 24: 1388–1390 180 Dorsal scapular nerve Genetic testing NCV/EMG Laboratory Imaging Biopsy +– Fig. This nerve is purely motor, and innervates the levator scapulae and rhomboid muscles (Fig. Function: To elevate and adduct the medial border of the shoulder blade (together with the rhomboid muscles). Almost no symptoms are reported, and usually only with powerful arm move- Symptoms ments.
FACE AND NECK REJUVENATION WITH MESOLIFT Aging accutane 10 mg line, sagging buy 40mg accutane overnight delivery, and wrinkling of the skin occur from accumulation of fat accutane 10 mg cheap, loss of skin elas- ticity cheap 10 mg accutane free shipping, and excessive free-radical damage. Using antioxidants and amino acids, mesother- apy can reduce fat from under the neck, decrease free radical damage, and tighten loose skin. Mesotherapy effects include the rejuvenation of face, eyelids, and neck, but only when performed along with a comprehensive treatment including skin care, use of ﬁllers, toxins, threads, and exfoliation (10). How many treatments are required before one sees results? Some patients require four to ﬁve treatments before beginning to see results while others may need more (12–17). MESOTHERAPY FOR CELLULITE & 271 FOUR ESSENTIAL QUESTIONS THAT EXPLAIN THE MESOTHERAPY TECHNIQUE & WHAT IS MESOTHERAPY? Mesotherapy consists of the introduction of drugs into the superﬁcial subcutaneous skin (46). The injections use minimal amounts of drugs as a complement to routine clinical procedures (47,48). The amount of the injection is determined by the proximity of the injection site to the site of the pathology. The different theories that have been proposed to explain the activity mechanisms of mesotherapy are as follows: & Dr. Pistor talks about reﬂex theory—the interruption of the visceral spinal tract when ID medication is administered (46). Bicheron talks about microcirculation stimulation (49). Dalloz Bourguinon believes the effect is due to activation of the microcirculatory, neuro-vegetative, and immunologic competing units (50). Didier Mrejen believes that all body organs have representation on the skin and has developed a skin map indicating their places of origin (8). Multedo says that superﬁcial administration of procaine produces a block in the Na–K pump, with the spread of medication through the extracellular space (9). Gancedo believes that when the administration is superﬁcial, there is greater spread and the effect is deeper. For better diffusion, the injections must be given at several points in parallel lines, without space in between. The depth of injection has to be 1 mm from the skin (10). Ballesteros has coined the phrase ‘‘energetic mesotherapy’’ (11,12). Kaplan combines multiple concepts and uses radiomarkers showing that the more superﬁcial the injection, the more extensive the diffusion (10). Drugs are injected into the skin in mesotherapy because treatment is applied at or closest to the disease. Drugs that are used intravenously, intramuscularly, subcutaneously, or intradermically are also suitable for use in mesotherapy (51). Therefore, drugs prepared in oily substances should not be administered, except those that have a content of propylene glycol in their formulation, which does not exceed a 20% concentration when diluted. All products must be water soluble, isotonic, and not cause nodules, abscesses, or necrosis at the site of injection. Because drugs are applied at the site of the pathologic condition, drug concentra- tions are higher in comparison to that obtained by other administration routes. Thus, greater therapeutic effects are achieved (52). The ID route is widely used by dermatologists for the administration of active drugs in speciﬁc disease states, for example, corticosteroids in the treatment of psoriasis. It is important to remember that the introduction of medicines intradermically confers properties that are speciﬁc to this form of administra- tion and that, beside the pharmacological actions pertaining to the active agents, other unforeseen effects may be observed, as well as the retardation and extension of the dose–effect relationship. One of the most transcendental aspects of these methodologies is the selection cri- teria of the drugs and their combinations; consequently, there are ten commandments to be followed when making this choice; i.
James Russel who also received neuropathological help from Dr buy accutane 20 mg with visa. Grisold and Zifko and reflect a large series of photographic clinical documentation accutane 10mg on line, that was accumu- lated over the years generic accutane 40 mg mastercard. We are aware that for many entities like polyneuropathies buy generic accutane 30mg on line, myopathies, and mononeuropathies several excellent monographs and teaching books have been written. However we found no other book which provides a complete overview in a structured and easily comprehensive pattern supported by figures and pictures. While writing for this book the authors have had fruitful discussions about several disease entities with individuals from the different schools of diagnosis, treatment and teaching in the US and in Europe. We hope that this book will be of clinical help for all physicians working with patients with neuromuscular disease. Zifko 5 Tools 7 Several important diagnostic tools are necessary for the proper evaluation of a patient with a suspected neuromuscular disorder. Each individual chapter in this book is headed by a “tool bar”, indicating the usefulness of various diagnostic tests for the particular condition discussed in the chapter. For example, genetic testing is necessary for the diagnosis of hereditary neuropathy and hereditary myopathy, while nerve conduction velocity (NCV) and elec- tromyography (EMG) can be important but are less specific for these diseases. Conversely, NCV and EMG are the predominate diagnostic tools for a local entrapment neuropathy like carpal tunnel syndrome. Some conditions will require autonomic testing or laboratory tests. The evaluation of a patient with neuromuscular disease includes a thorough The patient with history of the symptoms, duration of the present illness, past medical history, neuromuscular social history, family history, and details about the patient’s occupation, behav- disease iors, and habits. Much can be learned from the distribution of the symptoms and their temporal development. The types of symptoms (motor, sensory, autonomic, and pain) need to be addressed in detail. The history is followed by a clinical examination, which will assess signs of muscle weakness, reflex and sensory abnormalities, and autonomic changes, as well as give information about pain and impairment. The clinical examination is of utmost importance for several reasons. The findings will correlate with the patient’s symptoms, and the distribution of the signs (e. Documentation of the course of signs and symptoms will be useful in monitoring disease progression, and may guide therapeutic decisions. Documentation of the progression of neuromuscular disease (especially chronic diseases) should not be limited to changes measured by the ancillary tests described later in this section. Depending upon the disease, measurement of muscle strength, sensory measurements (e. Digital imaging, video clips, and photographs of patients provide a precise documentation of the patient’s movement capabilities, but may not be possible due to legal, ethical, and other concerns for the patient. The diagnostic hypothesis developed by the history and clinical exam can be confirmed by ancillary testing. Ancillary tests can also be used to monitor the stabilization or progression of the disease, and the impact of therapies. Standard electrophysiological tests include NCV, EMG, and repetitive nerve stimulation. Laboratory tests, such as creatine kinase, electrolyte assessment, and antibody testing (e. Genetic testing has become an important tool in the last twenty years, and can be used in many diseases to confirm a precise diagnosis. Some other tests, like autonomic testing (such as the Ewing battery and others) and quantitative sensory testing may not be available in some areas. MRI can be used to assess muscle inflammation and atrophy, and compression or swelling of peripheral nerves. The following description of diagnostic tools is intended to be a brief overview, with references for further reading. A peripheral nerve consists of bundles of axons surrounded by and embedded in a collagen matrix. The outer connective tissue covering is called the epineurium.
Deprived of oxygen cheap accutane 10mg with mastercard, which is VENTRICLES Of the four ventricles discount 5mg accutane free shipping, comparatively large spaces carried by blood accutane 5mg line, nerve cells in the a∑ected area cannot func- ﬁlled with cerebrospinal ﬂuid buy cheap accutane 10 mg line, three are located in the fore- tion and die. Thus, the part of the body controlled by those brain and one in the brainstem. The lateral ventricles, the two cells cannot function either. Stroke can result in loss of con- largest, are symmetrically placed above the brainstem, one in sciousness and death. WERNICKE’S AREA A brain region responsible for the compre- hension of language and the production of meaningful speech. Acetylcholine 4 Down syndrome 40–41 Magnetoencephalography Pons 3, 23 Action potential 4 Drug reward system 34 (MEG) 44 Positron emission tomography Addiction 33–36 Endocrine system 6–7, 25–27 Marijuana 36 (PET) 19, 43 Aging 28–29 Endorphins 6, 17 Memory 18–19 Primary visual cortex 12 and intellectual capacity 29 Epilepsy 31–32 Methylprednisolone 39 Procedural knowledge 18 AIDS 40 Epinephrine 25–26 Midbrain 3, 8 Prostaglandins 17, 31 Alcohol 34–36 Estrogen 7 Mitochondria 45 Psychostimulants 34–35 Alpha motor neurons 20 Fetal alcohol syndrome 35 Monoamine oxidase inhibitors Receptive ﬁeld 12 Alzheimer’s disease 36–37 Firing of neurons 4–5 (MAOIs) 32 Receptors 4 Amino acid transmitters 4–5 Flexion withdrawal 20–21 Morphine 6, 30, 31, 34 Reﬂex 20–21 Amphetamines 34 Fluoxetine 32 Motor cortex 3, 20 Regeneration 46 Amyloid protein 36–37 Forebrain 3 Motor neuron 20 Reproduction 7 Amyotrophic lateral sclerosis Functional Magnetic Reso- Motor unit 20 Schizophrenia 39–40 (ALS) 42 nance Imaging (fMRI) 44 Movement 20–21 Second messengers 7 Analgesia 30 Gamma-amino butyric acid MPTP 30 Selye, Hans 25 Androgen 7 (GABA) 5, 24, 32, 35 Multiple sclerosis 40 Serotonin 6, 32 Anxiety disorders 39 Gamma motor neurons 20 Myasthenia gravis 4 Single photon emission Autoimmune response 27 Gene 45 Myelin 4–5 computed tomography Autonomic nervous system diagnosis 44–45 Narcolepsy 24 (SPECT) 43 11, 25 therapy 46–47 Nerve growth factor (NGF) 46 Sleep 22–24 Axon 4–5 Glucocorticoids 7, 26–27 Nerve impulse 4, 5 REM sleep 22–24 Basal ganglia 19, 21, 30 Glutamate 5, 36, 38 Neuroﬁbrillary tangles 36 stages 22 Biological clock 7, 27 Hearing 14–15 Neurological trauma 38–39 disorders 23–24 Brain Heroin 34 Neuron 4–5 Smell 15–16 aging 28–29 Hippocampus 3, 18–19, 27 birth 9–10 Spinal cord 6, 11, 17, 20–21, anatomical organization 3 Huntington’s disease 41 migration 9–10 38–39, 46 development 8–11 Hypothalamus 3, 7, 24, 32 pathﬁnding 10 Strabismus 14 diseases 2–3 Immune system 27 survival 10–11 Stress 25–27 tumors 42 Information processing, Neurotransmitters 4–7 in arousal 25–26 Broca’s area 19 and hearing 14–15 Nicotine 33–34 chronic 27 Catecholamines 6 and learning and memory NMDA receptors 5, 18 and endocrine system 25–27 Central nervous system 6, 11 18–19 Norepinephrine 6 and schizophrenia 39 Cerebellum 19, 21 and movement 20–21 Obsessive-compulsive Stroke 37–38 Cerebral cortex and pain 16–17 disorder 39 Substance P 6 3, 17, 19, 23, 31 and taste and smell 15–16 Occipital lobe 3, 12 Synapse 4, 5, 29 Club drugs 36 and vision 12–13 Olfactory bulbs 15–16 Taste 15–16 Cocaine 34–35 Inhibitory neurons 20–21 Opiates 34–35 Temporal lobe 3, 18 Cortisol 25–26 Ion channels 4 Pain 16–17, 30–31 Testosterone 7 Costs of brain diseases 2–3 Language 19 Panic disorder 39 Thalamus 3 Crossed extension reﬂex 20–21 Learning 18–19 Parietal lobe 3 Touch 16–17 Declarative knowledge 18 Learning disorders 37 Parkinson’s disease 30, Tourette syndrome 41–42 Dementia 28, 36 Levodopa 6, 30 46–47 Tricyclic antidepressants 32 Dendrite 4–5 Limbic system 15 Peptides 6 Trophic factors 6, 46 Depression Long-term potentiation 18 Peripheral nervous system 11 Vision 12, 13–15 major 32 Lou Gehrig’s disease 42 Phenytoin 31 Wernicke’s area 19 manic 32 Magnetic resonance imaging Phobias 39 Working memory 18 Dopamine 6, 30, 34 (MRI) 43–44 Pituitary gland 6, 7, 32 53 Copyright © 2002 The Society for Neuroscience 11 Dupont Circle, NW, Suite 500 Washington, DC 20036 USA Telephone (202) 462-6688 www. No portion of this publication may be reproduced, stored in a retrieval system, or transmitted in any form or by any means, electronic, mechanical, photocopying, recording, or otherwise without permission of the The Society for Neuroscience. To acquire additional copies of this book, please visit our Web site www. The Society for Neuroscience Editor: Joseph Carey, Senior Director, Communications & Public Affairs Science writer: Leah Ariniello Researcher: Mary McComb Produced by Meadows Design Office Incorporated, Washington, DC www. Kibiuk, Baltimore, Maryland Printed and bound in China by Everbest Printing Company Fourth edition 06 05 04 03 02 5 4 3 2 1 isbn 0-916110-00-1 ISBN 0-916110-00-1 . No part of this publication may be reproduced in any material form (including photocopying or storing it in any medium by electronic means and whether or not transiently or incidentally to some other use of this publication) without the written permission of the copyright owner except in accordance with the provisions of the Copyright, Designs and Patents Act 1988 or under the terms of a licence issued by the Copyright Licensing Agency Ltd, 90 Tottenham Court Road, London, England W1P 9HE. Applications for the copyright owner’s written permission to reproduce any part of this publication should be addressed to the publisher. Warning: The doing of an unauthorised act in relation to a copyright work may result in both a civil claim for damages and criminal prosecution. The right of Peter Burke to be identified as author of this work has been asserted by him in accordance with the Copyright, Designs and Patents Act 1988. First published in the United Kingdom in 2004 by Jessica Kingsley Publishers Ltd 116 Pentonville Road London N1 9JB, England and 29 West 35th Street, 10th fl. In a discussion about nothing in particular, one comment hit me with its crystal certainty. At the age of 10 my daughter reassured me about my disabled son’s future in this way. She said: ‘Don’t worry daddy, when you are too old I will look after Marc. He has a condition referred to as spastic quadriplegia, and severe learning disabilities. These labels do not really represent Marc as we know him, but it helps with the image of his dependency and the reason why his sister understood that his care needs were in many ways different from her own. My daughter’s comment made me realise that it was not only I who was aware of my son’s disabilities, but my daughter also, and she was thinking of his future at a time when my partner and I were ‘taking a day at a time’. The inspiration drawn from that comment helped formulate a plan of research into the needs of siblings, and subse- quently this book. The book is structured to inform the practitioners (whether they are from the health, welfare or educational sectors), of the needs of siblings. I trust too, that the views expressed, based as they are on the experience of others and with some insights drawn from personal experience, will resonate with families in situations similar to my own. Outline of chapters Throughout the text quotations from families will be used to clarify points and issues raised, and detailed case examples will show how siblings react 9 10 / BROTHERS AND SISTERS OF CHILDREN WITH DISABILITIES to the experience of living with a disabled brother or sister, creating ‘disability by association’. Chapter 1 provides an introduction and a theoretical framework for analysis linking to the key concepts: inclusion, neglect, transitions and adjustments, children’s rights and finding a role for the practitioner. Models of disability are discussed to illustrate some of the differences found between professions. Chapter 2 introduces, in Part 1, a theoretically informed research typology (Table 2. The impact of disability on the family and siblings introduces some of the difference between parental perceptions and sibling expectations.
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